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Newly Discovered Bone Stem Cell Causes Premature Skull Fusion

Newly Discovered Bone Stem Cell Causes Premature Skull Fusion

The mechanobiology of craniosynostosis in Crouzon syndrome

Spatial transcriptomics reveals a role for sensory nerves in preserving cranial suture patency through modulation of BMP/TGF-β signaling

Frontiers FGF signaling in cranial suture development and related diseases

Inflammatory, Metabolic, and Genetic Mechanisms of Vascular Calcification

Newly Discovered Bone Stem Cell Causes Premature Skull Fusion, Newsroom

Functionalized 3D Hydroxyapatite Scaffold by Fusion Peptides-Mediated Small Extracellular Vesicles of Stem Cells for Bone Tissue Regeneration

Pharmaceutics, Free Full-Text

Molecular Mechanisms Involved in Craniosynostosis

Tricarboxylic Acid Cycle Metabolite-Coordinated Biohydrogels Augment Cranial Bone Regeneration Through Neutrophil-Stimulated Mesenchymal Stem Cell Recruitment and Histone Acetylation-Mediated Osteogenesis

Identification of stiffness-induced signalling mechanisms in cells from patent and fused sutures associated with craniosynostosis

Structural brain differences in school-age children with and without single-suture craniosynostosis in: Journal of Neurosurgery: Pediatrics Volume 19 Issue 4 (2017) Journals

The Biology of the Sutures of the Skull

Tissue scale properties of the extracellular matrix regulates nuclear shape, organisation and fate in the embryonic midline sutures